Good Omens: Lockdown and Crowley not mentioning his living situation in S2*
*till S2E6 when he asks if he can have his apartment back bc he's bored of living in his car but Aziraphale doesn’t hear bc mentally he’s in Alpha Centauri.
Having read the 'Crowley doesn't tell him' Neil Gaiman ask close to when I first listened to Lockdown (I lived under a rock until recently), my initial thought was HAS HE BEEN LIVING IN HIS CAR FOR YEARS?! but I think he was still in his apartment in 2020:
as far as Hell knows, Crowley just had a pool party in holy water (the holiest) so the higher-ups are probably willing to give him some space (plus Beelzebub is busy going on pub dates w Gabriel)
while there should be ~8 months between the end of Season 1 events (The Very First Day of the Rest of Their Lives on Sunday, Aug 25, 2019) and the Lockdown phonecall (on or near the 30 year anniversary on May 1, 2020), I can't imagine that's a very long time for Hell, especially if you're understaffed and busy dealing with fallout from Almostgeddon / going on pub dates
Shax dropping off mail and asking about the boiler seems like something one does in the first few months of living somewhere, not ~3 years in (if S2 is in 2023)
That said, I think the phone call underlines why Crowley never directly tells Aziraphale that he is living in the Bentley in S2, and it's just a great conversation (all hail Gaiman) sooo I wrote about it:
***Note: This post analyzes the Lockdown phonecall from Crowley's perspective only. Our heroine is feeling quite emotionally vulnerable at this point in time so things are going to hit him harder than they normally would.
I do not think Aziraphale meant to cause him pain (!!) but Crowley can't see that yet and I've written this post in a way that reflects that missing insight. (I explain in more detail in this reblog if you are interested) I am working on a companion post for Aziraphale's side of this conversation and how I think it affects his behavior in S2 because if we know anything about these two, it's that their exactlys are different exactlys.***
Crowley’s habit of sleeping to skip time like an RPG character by a campfire amuses me to no end, but in this context it feels heavy. Crowley already worries about losing time with what he loves and he probably hoped things would be different between him and Aziraphale after the events of S1. But things don’t change much. Then lockdowns start, and Crowley is trapped in his apartment alone, transcendentally bored, and unable to make his brain shut up. Sleeping a month away starts to sound less awful.
But Crowley hasn’t given up yet; he’s still awake when Aziraphale calls, and he’s even giving it two more days. Was he waiting for Aziraphale to call? Is it even possible not to at least kind of wait for someone’s call when you are cut off from everything and the caller has been your only friend and crush for millennia?
Aziraphale asks why Crowley isn't "out and about" tempting people or setting a bad example and he responds:
C: Everyone's so miserable and cooped up right now anyway, and I just… well… don't have the heart for it.
A: *glowing audibly* I'm not miserable~
C: Really?
Crowley sounds genuinely surprised at Aziraphale's happiness and quickly assumes it's because the angel has been around people. He's so lonely/depressed/in his own head that he hadn't even considered someone enjoying being 'cooped up'. *sob*
Aziraphale goes No actually I put the closed sign up in the window and I'm having the Time of My Life, never had so few customers, not in 200 years!, etc. Although, he says:
A: …There were a few young lads a couple of nights ago who broke in through the back and tried to steal the cashbox! But they soon saw the error of their ways~
C: *clearly amused* Did you smite them with your wroth?
A: Well I certainly gave them a good talking to, and I sent each of them home with cake~
C: *annoyed, swooning* Cake?
A: Quite a lot of cake, actually.
C: *physically ill from having such a giant crush on this dumbass baker/security guard* eeeekkkgghhh I'm gonna regret asking but.. ...rrgh..
*30 seconds of Aziraphale joyfully describing his baking while Crowley probably tries very hard not to imagine the angel eating each item in sensual slow motion* I stg you can hear him struggling in the background once or twice
A: …And once I've baked them, I have to eat them all myself, which was why I was so delighted—
C: To send your burglars home laden with baked goods, yes, nnyeaayeah I follow…
Crowley interrupts, finishing Aziraphale's sentence in his nervous hurry to say the next bit:
C: *loud inhale* You know, I could.. hunker down at your place. … Slither over and watch you eat cake. I could bring a bottle--a case of… something… drinkable…?
He's trying to sound so casual about it but this is someone who was rejected/abandoned by actual literal God after asking what he thought were welcome, uncontroversial questions. Asking makes him vulnerable. He's supposed to be the rescuer, not a demon in distress. He does not feel casual about asking.
Crowley knows it's unlikely but he's so miserable and desperate for company that he can't help but ask, just in case. Even the smallest chance of spending time trapped indoors with Aziraphale—with nothing to do but drink, watch him eat, and talk about things they'd normally avoid—is too tempting.
A: *panicking* Oh I— I— I— I— I'm afraid that would be Breaking All The Rules! *nervous breathing* Out of the question! I'll see you… when this is over.
C: Right. gnnehh. I'm setting the alarm clock for July. Good night, angel.
*dial tone*
And just like that, Crowley doesn't need two days to decide. The depression nap doubles in length. He doesn't hear how badly Aziraphale wants to say yes behind the fear, or maybe he does and it hurts worse because why isn't Crowley enough for him? You can almost hear the spiralling:
SHOCKING, asking made it worse. It always does doesn’t it? Why even bother? you just embarrass yourself.. SLITHER over? why did I say that *grumble grumble* of COURSE His Holy Holiness, your only friend in the universe, would rather eat cake by himself while everything goes to shit than ~deign~ to have you in his presence. "AsK aND yE sHaLl ReCeIvE" bugger this for a lark im going to bed
(a bit dramatic but we've all been there)
I imagine sleep doesn't come right away. Maybe his thoughts drift to when he sat beside the angel at a dark Tadfield bus stop after a rather eventful Saturday. Crowley must've felt a tiny bit hopeful when he invited Aziraphale to stay with him: Heaven had withdrawn its favor and the bookshop was gone; Aziraphale was like him now. Didn't that mean things would change?
"I don't think my side would like that." Apparently not.
In the end, Aziraphale did ride the bus back to Crowley's apartment and stayed till the next morning when he caught a cab, but only to sell the illusion. Crowley understood that as far as sides went, the angel was still on Heaven's, even if Heaven wasn't on his.
And now this: the entire world is shut down; there is nothing for Aziraphale to do but stay in and read and bake in his magically reconstituted bookshop and he still won't invite Crowley in. Burglars and un-fallen angels only—nobody who asks questions.
So... of course Crowley doesn't tell Aziraphale when he loses his apartment. He already knows what answer he would get; the angel has told him so many times. Aziraphale is a company man first, a companion to one very sad owl when convenient.
If Crowley works up the courage to say 'please take me in, I have nowhere else to go' and Aziraphale goes 'sorry, no, far too political, but I WILL risk being erased from the Book of Life to protect this nude amnesiac former coworker who always hated me,' it's going to be too much. You can't sleep long enough for that type of hurt to go away. Better not to say anything.
"Then nothing has to change, does it?"
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PSA: Covid Effects and Complications
Alright fekkers this is how we’re doing public health announcements now that society is collapsing :))
This isn’t a post to tell you to avoid it, but to ask you to read the headings and make sure you make an informed decision if you wanna go out and get infected. If you do take risks, remember to avoid very young/old/pregnant/unwell/vulnerable people, test regularly and wear a mask (yes, they work).
Covid is Not Just A Respiratory Illness
COVID-19 routinely affects organs throughout the body, not just the respiratory system, including the brain, heart, liver, GI tract, endocrine system and skin (Gupta et al., 2020)
Covid can be seen as a blood clotting disorder masquerading as a respiratory illness. (Janardhan et al., 2020)
Covid Causes Brain Damage
Mild Covid infection shows significant orbitofrontal cortical atrophy (shrinking of parts of the brain) and cognitive decline (Crunfli et al., 2022)
Mild COVID-19 infection can cause impaired neurogenesis (nervous tissue growth), myelin and oligodendrocyte (nerve insulation) loss and increased neurotoxic molecules around the central nervous system (Fernández-Castañeda et al., 2022 *preprint)
MRIs of 401 patients done before and after Covid infection showed reduction in global brain size, grey matter loss (orbitofrontal cortex and parahippocampal gyrus) and cognitive decline. (Douaud et al., 2022)
COVID increases the risk of neurodegenerative disorders- Alzheimer’s disease risk is 3.5x increased; Parkinson’s disease risk is 2.6x increased; ischaemic stroke risk is 2.7x increased and intracerebral haemorrhage (bleeding in the brain) risk is 4.8x increased. (Zarifkar et al., 2022)
Risks of cognitive deficit, dementia, psychotic disorders, and epilepsy or seizures are increased for at least 2 years following Covid infection. (Taquet et al., 2022)
In 25% of mild Covid cases, visuocontructive cognitive deficits are seen, associated with changes in brain structure and metabolism. (de Paula et al., 2022)
Hospitalised Covid patients cognitive loss is similar on average to that sustained with 20 years ageing, and equivalent to losing 10 IQ points. (Hampshire et al., 2022)
People reportedly recovered from Covid show decreased intelligence and significant cognitive deficits. (Hampshire et al., 2021)
Covid Causes Alzheimer’s-like Pathology and Accelerates Existing Alzheimer’s Disease
Covid invades cognitive centers of the brain and induces Alzheimer’s-like neuropathology (Shen et al., 2022)
Covid is associated with accelerated progression of Alzheimer’s disease (aaic.alz.org)
Covid produces proteins that form cytotoxic aggregates which damage neuronal cells, which parallels Alzheimer’s disease mechanism (Charnley et al., 2022)
COVID increases the risk of neurodegenerative disorders- Alzheimer’s disease risk is 3.5x increased; Parkinson’s disease risk is 2.6x increased; ischaemic stroke risk is 2.7x increased and intracerebral haemorrhage (bleeding in the brain) risk is 4.8x increased. (Zarifkar et al., 2022)
Risks of cognitive deficit, dementia, psychotic disorders, and epilepsy or seizures are increased for at least 2 years following Covid infection. (Taquet et al., 2022)
Covid Causes Kidney Damage
Mild Covid infection is associated with increased risk of kidney damage (Bowe et al., 2021)
Covid infection triples risk of End Stage Kidney Disease, requiring dialysis or kidney transplant (Bowe et al., 2021)
Covid Causes Diabetes
Covid patients have a 40% increased risk of being diagnosed with diabetes after first infection (Xie et al., 2022)
Covid infection is associated with an 81% increase in Diabetes incidence for 12+ weeks following infection (Rezel-Potts et al., 2022)
Mild Covid infections increase risk of Type 2 Diabetes development (Rathmann et al., 2022)
Covid infection leads to an average of 42% increased risk of Type 1 Diabetes across all ages. Risk increases most in pediatric patients- by 584%. (Quedan et al., 2022)
Covid Causes Cardiovascular Illness
Covid infection, even when mild, substantially increases risk of cardiovascular illness up to at least 1 year later (Xie et al., 2022)
Capillary density (how many small blood vessels are present) is reduced by 41% in sufferers of Long Covid 18 months after Covid infection (Osiaevi et al., 2022)
Acute Covid infection results in 6x increase in cardiovascular diagnosis; 11x increase in pulmonary embolism (blood clot in lung); 6x increase in atrial arrhythmias (abnormal heartbeat); 5x increase in venous thromboses (blood clot in vein). (Rezel-Potts et al., 2022)
A spike protein found on Covid-19 virus particles uses the body’s immune response to damage and inflame heart muscle cells. (heart.org)
People with Covid exhibited increased risks and 12-month burdens of incident cardiovascular diseases, including cerebrovascular disorders, dysrhythmias, inflammatory heart disease, ischemic heart disease, heart failure, thromboembolic disease and other cardiac disorders. Risk were evident even in those without prior cardiovascular disease. (Xie et al., 2022)
Risk of stroke more than doubles even with mild or asymptomatic Covid infection. Median time of stroke is 2 months after Covid diagnosis. (Tu et al., 2021)
Covid infection increases heart attack risk by 3-8x and stroke risk by 3-6x (Katsoularis et al., 2021)
Covid infection increases risk of deep vein thrombosis, pulmonary embolism and bleeding in the months following acute illness (Katsoularis et al., 2022)
Long Covid is associated with presence of microclots throughout the body. (Pretorius et al., 2021)
Long Covid patients may face an increased risk of abnormal blood clotting. (uclh.nhs.uk)
Covid Accelerates Biological Ageing
Accelerated biological ageing is seen in Covid infection (Cao et al., 2022)
Covid Damages the Immune System
Covid causes T-cell exhaustion, meaning the immune system is less able to fight off pathogens (Loretelli et al., 2021)
Previous infection with earlier SARS2 strains can lead to impaired immune responses to Omicron (Reynolds et al., 2022)
Covid infects and kills T-lymphocytes (key cells of the immune system), causing low T-lymphocyte counts 1(Guan et al., 2020), 2(Shen et al., 2022)
Long Covid patients show reactivation of latent Epstein-Barr (can cause MS) and Varicella Zoster (can cause shingles and Ramsey Hunt syndrome) viruses (Klein et al., 2022 *preprint)
2.8% of Long Covid patients reported Varicella Zoster Virus reactivation, leading to shingles, following Covid infection. Primary risk factors for VZV reactivation are age and immunodeficiency. (Davis et al., 2021)
Covid infection causes immunodefiency in recovered patients by downregulating a specific protein on B Cells (a type of immune cell). (Jing et al., 2021)
Covid Causes the Body to Attack Itself (Autoimmunity)
Covid causes production of autoantibodies which target the immune system, vascular cells, coagulation factors and platelets, connective tissue, and organ systems, including lung, the central nervous system compartment, skin, gastrointestinal tract and other tissues. (Wang et al., 2021)
Asymptomatic Covid infection can lead to severe Ulcerative Colitis (an inflammatory bowel disease). (Mora et al., 2022)
Mild Covid infection can produce significant levels of autoantibodies for 7+ months. (Bhadelia et al., 2021)
Covid infection precedes new appearance of autoimmune and inflammatory diseases. (Galleoti and Bayry, 2020)
Covid infection linked to development of vasculitis, arthritis, lupus and sarcoidosis. (Gracia-Ramos et al., 2021)
Autoantibodies linked to Lupus, Rheumatoid Arthritis, Guillain-Barré syndrome, immune thrombocytopaenia and autoimmune haemolytic anaemia found in patients following Covid infection. (Moody et al., 2021)
In a group of non-hospitalised healthcare workers with Covid, 54% tested positive for autoantibodies- these targeted skin, smooth muscle, neutrophils (a type of white blood cell of the immune system) and gastric parietal cells (cells in the gut). (Richter et al., 2021)
Covid May Affect Both Male and Female Fertility
COVID-19 virus can be found and continues to replicate in the testes even after death (Costa et al., 2022 *preprint)
COVID-19 infects the testes and damages spermatogenesis (sperm production) (Ma et al., 2020)
Covid virus particles found in penis tissue of men infected 6-8 months earlier, who later experienced erectile dysfunction (Kresch et al., 2021)
Study shows total sperm number lower in men infected with Covid at 3 month follow up (Best et al., 2021)
Testes of Covid patients show significant seminiferous tubular injury and reduced Leydig cells- cells that produce testosterone. (Yang et al., 2020)
11 of 26 (42%) men with mild/moderate Covid infection showed incidental (asymptomatic) epididymitis on Doppler ultrasound - a condition that can cause infertility (Carneiro et al., 2021)
A case of premature ovarian failure due to Covid infection (Madaan et al., 2021)
Another case of premature ovarian insufficiency in a 34-year-old following Covid infection (Wilkins and Al-Inizi, 2021)
Ovarian injury, including declined ovarian reserve and reproductive endocrine disorder, can be observed in a study of women in China infected with Covid. (Ding et al., 2021)
Study finds men who had seemingly fully recovered from Covid infection developed decreased sperm count and motility and abnormally shaped sperm. (Ghosh et al., 2022)
Covid Causes Erectile Dysfunction
Covid virus particles found in penis tissue of men infected 6-8 months earlier, who later experienced erectile dysfunction (Kresch et al., 2021)
A Long Covid survey found 15% of men reported sexual dysfunction and 3% reported a decrease in genital size. (Davis et al., 2021)
Prevalence of erectile dysfunction 3x as common in men after Covid infection (28% vs 9% in controls) (Sansone et al., 2021)
Prevalence of erectile dysfunction in Thai men reported as 65% following Covid infection (Harirugsa et al., 2021)
Another study showing risk of erectile dysfunction triples following Covid infection. (Katz et al., 2021)
Study finds that Covid infection leads to 6-fold increased risk of erectile dysfunction, which worsens men’s mental health. (Hsieh et al., 2022)
Covid Causes Autonomic Nervous System Dysfunction
30% of 4000 Long Covid patients met the criteria for a diagnosis of Postural Orthostatic Tachycardia Syndrome, a type of dysautonomia (Davis et al., 2021)
Dysautonomia (autonomic nervous system dysfunction) may be responsible for fatigue and hypoxia in Long Covid patients. (Barizien et al., 2021)
Covid infection frequently causes abnormalities in autonomic nervous system tests, as well as worsening pre-existing dysfunction. Abnormalities included orthostatic intolerance, fainting, heachaches, burning pains, excessive sweating and lightheadedness. (Shouman et al., 2021)
67% of Long Covid patients have moderate-to-severe autonomic dysfunction, regardless of severity of initial Covid infection. (Larsen et al., 2022 *preprint)
Covid infection could result in gastric dysmotility and paralysis (stomach and intestines become unable to move food through). (Coles et al., 2022)
Covid Can Seriously Harm Children
SARS2 causes increased hospital admissions, mortality rate and absolute numbers of deaths in children, compared to Influenza (Shein et al., 2022)
Asymptomatic infection in children can lead to a serious, multiorgan hyperinflammatory syndrome (Riphagen et al., 2020)
An epidemic of hepatitis in healthy children could be linked to previous COVID-19 infection (science.org)
Pulmonary dysfunction persists even in children considered to be recovered from Covid (Heiss et al., 2022 *preprint)
Covid leads to a 3x increased risk of psychotic disorders in children (Taquet et al., 2022)
Children are twice as likely to develop epilepsy or seizures following Covid infection, compared to following other respiratory infections (Taquet et al., 2022)
Children are at an increased risk of epilepsy, encephalitis, nerve, nerve root and plexus disorders up to at least 2 years after Covid infection (Taquet et al., 2022)
Intracranial (brain) bacterial infections have increased during the Covid pandemic, occuring during or just after Covid infection. One Michigan children’s hospital reports a 236% increase. (Khuon et al., 2022)
235,000 children in England have Long Covid symptoms lasting 12+ weeks that affect their daily life (ONS.gov.uk)
21% of Year 13 pupils missed 4+ weeks of school due to Covid for the 21/22 academic year in England (suttontrust.com)
Children and teens who’ve had Covid are at greater risk for blood clots, heart problems, kidney failure, and Type 1 diabetes (Kompaniyets et al., 2022)
5.2 million children have lost a parent or caregiver to Covid infection. (Unwin et al., 2022)
Covid infection leads to an average of 42% increased risk of Type 1 Diabetes across all ages. Risk increases most in pediatric patients- by 584%. (Quedan et al., 2022)
Covid Can Endanger Pregnancy and the Growing Baby
Covid infection during pregnancy increases risk of preterm delivery (Edlow et al., 2022)
Covid infection at delivery increases risk of stillbirth (DeSisto et al., 2021)
Covid infection during pregnancy increases risk of neurodevelopmental disorder diagnosis in babies by 2.17x during first year of life (Edlow et al., 2022)
Risk of severe Covid infection is higher in pregnant women. (Rad et al., 2021)
Newborns born to mothers who had recovered from Covid 10+ weeks prior to birth show viral mRNA and proteins in their stool and signs of intestinal inflammation. (Jin et al., 2022)
Covid infection destroys the placenta, starving the baby of oxygen, resulting in increased risks of stillbirth and neonatal deaths. (Schwartz et al., 2022)
Covid Can Lead to Development of New Allergies
Mast cell activation syndrome (MCAS) may be triggered by Covid infection, resulting in new allergies and risk of anaphylaxis. (Afrin et al., 2020)
Mast cell activation symptoms are increased in Long Covid (Weinstock et al., 2021)
Covid Worsens Mental and Psychological Health
Covid diagnosis associated with increased risk of mental health diagnosis and neurocognitive decline (Xie et al., 2022)
Risks of cognitive deficit, dementia, psychotic disorders, and epilepsy or seizures are increased for at least 2 years following Covid infection. (Taquet et al., 2022)
Covid Reinfection is Common and Increasingly Detrimental to Health
Reinfection with Covid increases risk of hospitalization, death and long covid by more each time 1(Al-Aly et al., 2022 *preprint), 2(World Health Organisation)
Covid reinfections are common. Mean time between 1st and 2nd infection is 79 days, and between 2nd and 3rd infection is 65 days. (Al-Aly et al., 2022 *preprint)
Covid reinfection is possible as soon as 19 days after initial infection. (Ren et al., 2022)
Long Covid is Common, Serious and Potentially Disabling
1 in 5 (20-30%) develop a new health condition following Covid infection (Bull-Otterson et al. 2022)
Just below 1 in 10 (9.3%) triple vaccinated people are not recovered 4-8 weeks after Omicron infection in the UK (ONS.gov.uk)
1 in 7 (14%) of 11-18 year olds have symptoms 15 weeks after COVID-19 infection (Stephenson et al., 2021)
Long Covid causes disability and unemployment (theguardian.com)
Two million days of healthcare staff absences were lost to Long Covid during the first 18 months of the pandemic in England (the guardian.com)
On average, healthcare staff absent with Long Covid are off for more than 80 days in England (theguardian.com)
A US Long Covid group reports that 44% of those affected are out of work (longhauler-advocacy.org)
235,000 children in England have Long Covid symptoms lasting 12+ weeks that affect their daily life (ONS.gov.uk)
21% of Year 13 pupils missed 4+ weeks of school due to Covid for the 21/22 academic year in England (suttontrust.com)
The proportion of people unemployed and not seeking work due to Long Covid has doubled in the past year in the UK (theguardian.com)
Long Covid survey of nearly 4000 finds 45% required a reduced work schedule and 22% could not work due to illness (Davis et al., 2021)
88% of Long Covid sufferers experience cognitive dysfunction and memory problems (Davis et al., 2021)
80,000 people estimated to have left employment due to Long Covid by March 2022 in UK (Reuschke and Houston, 2022)
2.9 million people of working age in the UK have had, or currently have, Long Covid (Reuschke and Houston, 2022)
Long Covid has over 200 symptoms spanning 10 organ systems. (Davis et al., 2021)
Graph via @davidsteadson on Twitter
Vaccination Does Not Fully Prevent Long Covid
16% of Covid infections lead to Long Covid after 3 vaccinations (Azzolini et al., 2022)
Just below 1 in 10 (9.3%) triple vaccinated people are not recovered 4-8 weeks after Omicron infection in the UK (ONS.gov.uk)
Vaccination only reduces risk of Long Covid by 15%. (Al-Aly et al., 2022)
Covid Persists in the Body after Initial Infection
Covid can persist throughout the body and brain even following mild/asymptomatic infections, for at least 230 days (Chertow et al., 2021 *preprint)
COVID-19 can persist within the gut for at least 7 months after infection 1(Gaebler et al., 2021), 2(Natarajan et al., 2022)
Residual COVID-19 virus has been found in the appendix and breast tissue, 175- and 462-days post-infection, respectively (Goh et al., 2022 *preprint)
COVID-19 virus can persist in the eyes after initial infection (Armstrong et al., 2021)
COVID-19 virus can be found and continues to replicate in the testes even after death (Costa et al., 2022 *preprint)
Covid virus particles found in penis tissue of men infected 6-8 months earlier, who later experienced erectile dysfunction (Kresch et al., 2021)
Viral Persistence Can Cause Serious Illness Many Years Later
We do not know the long term effects of Covid Infection and Persistence.
Persistent Hepatitis C infection increases risk of Hepatocellular carcinoma (liver cancer) (Mitchell et al., 2015)
Persistent Human Papillomavirus (HPV) infection causes cervical cancer (Sudenga et al., 2013)
Persistent HIV infection leads to immunodeficiency and AIDS (Pauza, 1988)
Persistence of Epstein Barr Virus (EBV) can cause development of multiple sclerosis (Ruprecht, 2020)
Persistence of varicella zoster virus (VZV), which causes chickenpox, can result in shingles and Ramsey Hunt Syndrome (Gershon et al., 2015)
These effects of Covid are not easy to learn about, but it is essential that people know the risks. This is not fearmongering- it is not “what if”s and “maybe”s- these are events that are happening right now, around the world.
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