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#public health
panicinthestudio · 3 hours ago
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Further reading:
CBC: University of Alberta Hospital nearly doubles ICU beds as Edmonton zone hits 89% capacity, September 16, 2021
CBC: Calgary to take own measures to act on COVID-19 crisis, saying province 'doesn't care', September 16, 2021
@allthecanadianpolitics, @abpoli, @politicsofcanada
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rjzimmerman · a day ago
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Excerpt from this story from the New Scientist:
Wildfires akin to those that devastated parts of Greece, Siberia and North America this year are also taking an invisible but deadly toll on human health. The proportion of deaths linked to short-term exposure to smoke released by the fires is nearly as high as those from heatwaves, a new estimate suggests.
“This is a little bit of a surprise because wildfires are not very frequent. Smoke is a serious problem [for public health],” says Yuming Guo at Monash University in Melbourne, Australia.
Guo and his colleagues matched data on daily deaths from all causes across 749 cities in 43 countries between 2000 and 2016 against modelling of how exposed those people were to tiny particulates (PM2.5) released by wildfires. They linked 33,510 of 65.6 million total deaths a year to the wildfire pollution, or 0.62 per cent of all deaths, after adjusting for other possible explanations such as temperature. By contrast, heat-linked deaths are estimated to make up about 0.91 per cent of deaths.
Guatemala had the highest percentage of deaths linked to PM2.5 released by the fires, at 3.04 per cent, followed by Thailand, Paraguay, Mexico and Peru. The US had a relatively small percentage, at 0.26 per cent, as did Greece at 0.33 per cent, despite recent wildfires in these countries.
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themedicalstate · a month ago
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Escape
Miniature Art by Tatsuya Tanaka (Follow the artist here)
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mckitterick · 11 months ago
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COVID-19: Airborne Transmission
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COVID-19 Is Transmitted Through Aerosols. We Have Enough Evidence, Now It Is Time to Act
Finally, a practical article about coronavirus transmission and ways to mitigate its spread - including best practices for staying healthy. (These are just excerpts.)
Also, this is the first I've read verification of aerosol / airborne COVID transmission - hugely important information.
TIME magazine article (free): X
Two articles referenced in this piece:
Oxford Academic journal article (free): "It Is Time to Address Airborne Transmission of Coronavirus Disease 2019 (COVID-19)"
Science Direct article (free): "Short-range airborne route dominates exposure of respiratory infection during close contact"
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mednerds · a month ago
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A Story of Two Chest X-Rays
One patient with a vaccine and one patient without. Version 2—for the crowd that wants specifics without violating patient privacy (these are published cases).
The top picture is of a 47-year-old man who received the Pfizer vaccine and developed COVID19 2 weeks after. He was overweight (BMI = 29), but without any known comorbidities. He had a runny nose, mild body aches, mild cough. His chest X-ray is relatively normal.
The bottom picture is a 50-year-old active female patient without obesity and not on medications. Her chest X-ray shows diffuse opacities, consolidations in both lungs with lung damage (all the fluffy white), and a pattern that looks like the worst feared complication of COVID19—acute respiratory distress syndrome (ARDS). She needed intubation, mechanical ventilation, and ECMO (extra-corporal membrane oxygenation) – the most life support we can offer.
The mRNA vaccines are effective at preventing severe disease and death— even with the Delta variant. ICUs are starting to fill up with COVID19 patients again. This post was to emphasize that the vaccines are ~95% effective at preventing case 2 from happening. Of course, there are also mild cases of COVID in unvaccinated individuals.
Black lung tissue on the X-Ray is where the oxygen you breathe in gets into the blood. All the fluffy white is where the oxygen cannot get into your blood because of infection/inflammation, that’s essentially what ARDS is and the goal of mechanical ventilation/ECMO is to protect that damaged lung tissue and get the oxygen into your bloodstream and to your brain, heart and other organs.
Source: Jesse O’Shea M.D. Follow here: jesseosheamd.
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chismosite · 11 days ago
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a snapshot of the violence of U.S. housing policy at this moment of the pandemic (early sep. 2021)
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"Blackstone to Buy $7.3 Billion of AIG Housing, Insurance Assets"
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"California promised 100% rent forgiveness for struggling tenants. Most are still waiting"
U.S. economy transfers massive amount of wealth from poor to rich, triggered by the COVID-19 pandemic
6,200,000 people are behind on rent and at risk of eviction
State programs fail to stop evictions or cancel rent and are ineffective at distributing aid
The Supreme Court rules the CDC eviction moratorium unconstitutional
Private funds are buying homes en masse, including Affordable Housing
COVID-19 cases rise as evictions rise.
Every level of U.S. government, in collaboration with the private housing market, is set to kill off its poorest people to enforce its system of private housing
these points are only a handful out of the current state of housing. I can't stress how catastrophic it is and will grow to be. No amount of
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awesome-picz · a year ago
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Pics That Show How Differently Celebrities And Normal People Are Treated During The Coronavirus Outbreak
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themedicalstate · a month ago
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Your Vaccinated Immune System Is Ready for Breakthroughs
Getting COVID-19 when you’re vaccinated isn’t the same as getting COVID-19 when you’re unvaccinated.
A new dichotomy has begun dogging the pandemic discourse. With the rise of the über-transmissible Delta variant, experts are saying you’re either going to get vaccinated, or going to get the coronavirus.
For some people—a decent number of us, actually—it’s going to be both.
Coronavirus infections are happening among vaccinated people. They’re going to keep happening as long as the virus is with us, and we’re nowhere close to beating it. When a virus has so thoroughly infiltrated the human population, post-vaccination infections become an arithmetic inevitability. As much as we’d like to think otherwise, being vaccinated does not mean being done with SARS-CoV-2.
Post-vaccination infections, or breakthroughs, might occasionally turn symptomatic, but they aren’t shameful or aberrant. They also aren’t proof that the shots are failing. These cases are, on average, gentler and less symptomatic; faster-resolving, with less virus lingering—and, it appears, less likely to pass the pathogen on. The immunity offered by vaccines works in iterations and gradations, not absolutes. It does not make a person completely impervious to infection. It also does not evaporate when a few microbes breach a body’s barriers. A breakthrough, despite what it might seem, does not cause our defenses to crumble or even break; it does not erase the protection that’s already been built. Rather than setting up fragile and penetrable shields, vaccines reinforce the defenses we already have, so that we can encounter the virus safely and potentially build further upon that protection.
To understand the anatomy of a breakthrough case, it’s helpful to think of the human body as a castle. Deepta Bhattacharya, an immunologist at the University of Arizona, compares immunization to reinforcing such a stronghold against assault.
Without vaccination, the castle’s defenders have no idea an attack is coming. They might have stationed a few aggressive guard dogs outside, but these mutts aren’t terribly discerning: They’re the system’s innate defenders, fast-acting and brutal, but short-lived and woefully imprecise. They’ll sink their teeth into anything they don’t recognize, and are easily duped by stealthier invaders. If only quarrelsome canines stand between the virus and the castle’s treasures, that’s a pretty flimsy first line of defense. But it’s essentially the situation that many uninoculated people are in. Other fighters, who operate with more precision and punch—the body’s adaptive cells—will eventually be roused. Without prior warning, though, they’ll come out in full force only after a weeks-long delay, by which time the virus may have run roughshod over everything it can. At that point, the fight may, quite literally, be at a fever pitch, fueling worsening symptoms.
Vaccination completely rewrites the beginning, middle, and end of this story. COVID-19 shots act as confidential informants, who pass around intel on the pathogen within the castle walls. With that info, defensive cells can patrol the building’s borders, keeping an eye out for a now-familiar foe. When the virus attempts to force its way in, it will hit “backup layer after backup layer” of defense, Bhattacharya said.
Prepped by a vaccine, immune reinforcements will be marshaled to the fore much faster—within days of an invasion, sometimes much less. Adaptive cells called B cells, which produce antibodies, and T cells, which kill virus-infected cells, will have had time to study the pathogen’s features, and sharpen their weapons against it. While the guard dogs are pouncing, archers trained to recognize the virus will be shooting it down; the few microbes that make their way deeper inside will be gutted by sword-wielding assassins lurking in the shadows. “Each stage it has to get past takes a bigger chunk out” of the virus, Bhattacharya said. Even if a couple particles eke past every hurdle, their ranks are fewer, weaker, and less damaging.
In the best-case scenario, the virus might even be instantly sniped at by immune cells and antibodies, still amped up from the vaccine’s recent visit, preventing any infection from being established at all. But expecting this of our shots every time isn’t reasonable (and, in fact, wasn’t the goal set for any COVID-19 vaccine). Some people’s immune cells might have slow reflexes and keep their weapons holstered for too long; that will be especially true among the elderly and immunocompromised—their fighters will still rally, just to a lesser extent.
Changes on the virus side could tip the scales as well. Like invaders in disguise, wily variants might evade detection by certain antibodies. Even readily recognizable versions of the coronavirus can overwhelm the immune system’s early cavalcade if they raid the premises in high-enough numbers—via, for instance, an intense and prolonged exposure event.
With so many factors at play, it’s not hard to see how a few viral particles might still hit their mark. But a body under siege isn’t going to throw its hands up in defeat. “People tend to think of this as yes or no—if I got vaccinated, I should not get any symptoms; I should be completely protected,” Laura Su, an immunologist at the University of Pennsylvania, said. “But there’s way more nuance than that.” Even as the virus is raising a ruckus, immune cells and molecules will be attempting to hold their ground, regain their edge, and knock the pathogen back down. Those late-arriving efforts might not halt an infection entirely, but they will still curb the pathogen’s opportunities to move throughout the body, cause symptoms, and spread to someone else. The inhospitality of the vaccinated body to SARS-CoV-2 is what’s given many researchers hope that long COVID, too, will be rarer among the immunized, though that connection is still being explored.
Breakthroughs, especially symptomatic ones, are still uncommon, as a proportion of immunized people. But by sheer number, “the more people get vaccinated, the more you will see these breakthrough infections,” Juliet Morrison, a virologist at UC Riverside, said. (Don’t forget that a small fraction of millions of people is still a lot of people—and in communities where a majority of people are vaccinated, most of the positive tests could be for shot recipients.) Reports of these cases shouldn’t be alarming, especially when we drill down on what’s happening qualitatively. A castle raid is worse if its inhabitants are slaughtered and all its jewels stolen; with vaccines in place, those cases are rare—many of them are getting replaced with lighter thefts, wherein the virus has time only to land a couple of punches before it’s booted out the door. Sure, vaccines would be “better” if they erected impenetrable force fields around every fortress. They don’t, though. Nothing does. And our shots shouldn’t be faulted for failing to live up to an impossible standard—one that obscures what they are able to accomplish. A breached stronghold is not necessarily a defeated stronghold; any castle that arms itself in advance will be in a better position than it was before.
There’s a potential silver lining to breakthroughs as well. By definition, these infections occur in immune systems that already recognize the virus and can learn from it again. Each subsequent encounter with SARS-CoV-2 might effectively remind the body that the pathogen’s threat still looms, coaxing cells into reinvigorating their defenses and sharpening their coronavirus-detecting skills, and prolonging the duration of protection. Some of that familiarity might ebb with certain variants. But in broad strokes, a post-inoculation infection can be “like a booster for the vaccine,” Su, of the University of Pennsylvania, said. It’s not unlike keeping veteran fighters on retainer: After the dust has settled, the battle’s survivors will be on a sharper lookout for the next assault. That’s certainly no reason to seek out infection. But should such a mishap occur, there’s a good chance that “continuously training immune cells can be a really good thing,” Nicole Baumgarth, an immunologist at UC Davis, said. (Vaccination, by the way, might mobilize stronger protection than natural infection, and it’s less dangerous to boot.)
We can’t control how SARS-CoV-2 evolves. But how disease manifests depends on both host and pathogen; vaccination hands a lot of the control over that narrative back to us. Understanding breakthroughs requires some intimacy with immunology, but also familiarity with the realities of a virus that will be with us long-term, one that we will probably all encounter at some point. The choice isn’t about getting vaccinated or getting infected. It’s about bolstering our defenses so that we are ready to fight an infection from the best position possible—with our defensive wits about us, and well-armored bodies in tow.
By Katherine J. Wu (The Atlantic). Illustration by Adam Maida/The Atlantic.
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mednerds · a month ago
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Get Vaccinated 
Right now, most people admitted to hospitals for COVID19 are unvaccinated. COVID-19 vaccines help protect you from severe illness, being admitted to the hospital, and death.
If you’re not vaccinated against COVID-19, get vaccinated as soon as you can: www.vaccines.gov.
Source: Centers for Disease Control and Prevention updates from Aug. 6, 2021.
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Foods of Health: Plant-Based
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There could really be a lot of things to be impressed of when it comes to the potential health benefits associated with healthcare. Taking care of one's self could be one of the basic things one might need to do with himself. Plant-based options have recently stormed the social media platforms and it could be great.
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Being knowledgeable enough about what's in it for you could be an advantage one could want to have. Being vegan could be good to some people, but just starting to have a more plant-based alternatives at an ample level could be a good start for every people. Care to see what your choices could be? A good list of those could be found in here
Check disclaimer on profile and landing page
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digital-medic · 8 months ago
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This was written several months before the Capital insurrection.
You can read the full article here [via Medium].
We are (and have been) in dangerous territory. Unless major structural reforms are implemented for both Congress and the executive branch, it's only a matter of time before we become a failed state.
The fetish of "American Exceptionalism" has anesthetized any sustained effort at reform in the belief that "we'll bounce back like we always do". Meanwhile our healthcare, employment, civil and political institutions are crumbling at a rapid pace leaving families hungry and desperate, while a handful of CEOS pay less and control more.
How bad do things have to get before we realize things are bad?
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