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livingwithmbc · 2 years

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Living with Metastatic Breast Cancer (MBC)

The past few years have been rocky to put it mildly, not just as a nation or planet, but also personally.

I'm creating this space to share periodic updates and glimpses into what it's like living with metastatic breast cancer (referred to as MBC going forward because I'm lazy and don't like typing it every time) and whatever else I feel like. I don't promise to post consistently, only as I find it helpful and have the time and energy. I do, however, promise to be real, honest, crass, and vulnerable about my experiences. I curse like a sailor and if that isn't for you, no hard feelings, but this may not be the space for you, and that's perfectly okay. I don't particularly enjoy writing, nor am I great at it, but I recognize its value and how cathartic it can be. I've always been a naturally private person as I enjoy my quiet life, but there's nothing private about having BC. Appointment after I'm appointment I remove my shirt and bra as it seems like just about every medical professional needs to feel my breast lump. Just as cancer has invaded my breast, medical traumas began invading my life. Privacy seems almost comical these days, and I was living in denial while thinking I could do this all on my own without needing the support of my friends and family. I was very wrong. I didn't (and still don't) want pity from others. Life never promised to be fair. We don't choose the cards life deals us, but it's up to us to play the hell out of those cards, and I've got a killer poker face. So ask me the questions and I'll respond when I can. This is not a journey in which it's helpful to go it alone and if anything, it's detrimental to try. Something else noteworthy is that I have ADHD (thanks, dad!). As someone with ADHD, my brain jumps around. A lot. This is evident when talking with me but also in my writing as well. Bear with me and welcome to the shitshow.

For those of you that don't know me well (or perhaps at all), I turned 33 last month and work as a mental health counselor in Indiana. I've been married to my saint of a spouse for just shy of a decade and he's been my rock. In 2020, I was gearing up to graduate with my master's in clinical mental health counseling with plans to begin our family shortly thereafter. Unfortunately, the universe had other plans.

During the summer of 2020, my spouse was diagnosed with non-Hodgkins Lymphoma at age 30, after being short of breath for no clear reason, and being gaslit by medical professionals for several months about not feeling well. To say this was a shock was an understatement. Only old people get cancer, I thought, not seemingly healthy and active 30-year-olds. Our plans to begin trying to conceive were temporarily tabled as the focus shifted to my husband's health. I was devastated but chanted the mantra, "this too shall pass". We were told we had to wait at least 2 years to try to have kids due to the intense medications and treatments he was on. He spent months doing aggressive rounds of chemo, all while working almost full time. To say he's my hero would be putting it mildly. It's been about 2 years since his diagnosis and I'm thrilled to report he's still in remission!

Fast forward to early 2022. I noticed some dimpling under one of my breasts, but genuinely didn't think too much of it. Historically speaking, I've never been an overly anxious person, and typically don't worry much until there's truly something to worry about. My spouse encouraged me to schedule an appointment ASAP (he's the worrier). I already had an OB appointment upcoming so I planned to discuss the dimpling then as my neurodivergent brain couldn't handle making more phone calls for appointments and things. Unfortunately, the doctor felt a lump (I couldn't) and the ensuing weeks and months would prove to be an overwhelming whirlwind of fears, appointments, and uncertainties.

I like to think I have a good sense of humor, even if it's dark (I'd argue you have to have dark humor to survive in the mental health field). On Friday the 13th of May I was told my breast biopsy confirmed the worst: I have invasive ductile carcinoma. Jason was nowhere in sight but I would have been more accepting of his existence than me having cancer. I'll never forget the look of pity on the nurse's face delivering that news. I could tell she was going out of her way to try and make me feel better about the diagnosis, saying things along the lines of, "it was caught early, you won't die. You'll be fine." I remember taking the news surprisingly well and not being too phased by it. "I'm going to kick cancer's ass," I thought. I'm stubbornly determined when I set my mind to a task and cancer was no different in my mind. Mind over matter, as they say. Hell, I was even given a BC swag bag on my way out the door. I quickly got scheduled with an oncologist who set up scans, blood draws, the whole gambit. Getting breast cancer at 32 was jarring for the medical providers around me given that I have no family history of breast cancer. Genetic testing was order and I learned that I have an ATM genetic mutation, pre-disposing me to breast cancer and a handful of other cancers. The results were bittersweet as it provided answers to the "why" of cancer early in life, but shifted the initial surgery treatment plan to opting for a double mastectomy. I was generally still in high spirits, and made light of it all, joking about getting a shiny new rack as a silver lining of a shitty situation. When life gives you lemons, make tittyaide, I said. As scan results began to roll in, the plan abruptly shifted. A suspicious spot was found on my sternum and a biopsy confirmed the worst: the cancer had already spread to my sternum, meaning I was now dealing with stage 4 metastatic breast cancer, a completely different beast than when BC is caught early. Surgery got cancelled and starting endocrine therapy ASAP was the new plan to try and shrink the tumors. I had no idea that multiple types of BC exist, all with different treatment implications. My specific type is ER/PR+, HER2-, meaning, my cancer feeds off my hormones. The treatment? Reduce the estrogen in my body as quickly as possible and transition me into menopause, thus stifling the cancer's fuel source. In all this scary news, the thing I mourned the deepest (and still do) is the uphill journey I will face to becoming a mom. Chemo made my spouse sterile and I am unable to carry a pregnancy as I cannot stop treatment long enough to sustain a pregnancy. People mean well when they offer comments like, "you can adopt!" but I'm here to tell you how painful and invalidating that response is. There is lifelong grief associated with infertility for those that want biological children. Even if we are able to pursue foster to adoption (the only "affordable" option to becoming a parent), I will always grieve not getting the experience of being pregnant and having biological kids. As cliché as it is, it's true that you don't always realize how badly you want something until it's no longer an option.

MBC, unlike early onset BC, is considered incurable. It's not an instant death sentence, but any doctor will let you know that it's essentially terminal, meaning it's a slow death. Living with MBC is a very, very different experience as there is no end in sight unlike many other cancer experiences. I will be in treatment for the rest of my life. The statistics for long-term survival aren't great, but I know I'm much more than a statistic. My goal is to live the most fulfilling life I can for as long as I can, and I hope that means I'll be around for a very long time. There's nothing like the threat of dying to make you appreciate each and every day, including the people in your life, the jobs, the pets, nature, etc. I believe maintaining a positive mindset while looking for learning opportunities is so important in overcoming any obstacles in life and I am so incredibly thankful for all those that have shown their love and support. If you read all of my ramblings, thank you for your patience. Take time to appreciate and express gratitude for the good things in your life. No matter how bad the circumstances may be, there is always something to be grateful for. <3

#breast cancer #metastatic breast cancer #nonhodgkins #lymphoma #atm #genetic mutation #hereditary cancer #infertility #compassion #medical trauma #mysogyny #adult adhd #gratitude #mental health #griefjourney #cancer #hereditary

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drforambhuta · 3 months

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The common types of hereditary cancer syndromes include:

1. Inherited Breast and Ovarian Cancer Syndrome (HBOC): HBOC primarily stems from mutations within the BRCA1 and BRCA2 genes. Females carrying these genetic alterations face notably heightened risks of breast and ovarian malignancies. Furthermore, BRCA mutations correlate with elevated susceptibilities to other cancers, such as prostate, pancreatic, and male breast cancer.

2. Lynch Syndrome (Hereditary Nonpolyposis Colorectal Cancer, HNPCC): Lynch syndrome arises from mutations affecting genes responsible for DNA mismatch repair, notably MLH1, MSH2, MSH6, and PMS2. Those with Lynch syndrome exhibit increased predispositions to colorectal cancer, alongside risks of endometrial, ovarian, gastric, and urinary tract cancers.

3. Familial Adenomatous Polyposis (FAP): FAP manifests with the proliferation of numerous polyps within the colon and rectum, which pose a cancer risk if untreated. Primarily linked to mutations in the APC gene, FAP substantially heightens the likelihood of early-onset colorectal cancer.

4. Li-Fraumeni Syndrome (LFS): LFS, a rare hereditary cancer syndrome, arises from mutations in the TP53 tumor suppressor gene. Individuals with LFS confront elevated lifetime risks of various cancers, including breast cancer, sarcomas, brain tumors, and adrenal gland tumors, often occurring at a young age.

5. Hereditary Diffuse Gastric Cancer (HDGC): HDGC entails an escalated susceptibility to diffuse gastric cancer, characterized by infiltration of the stomach wall. This predisposition predominantly results from mutations in the CDH1 gene, which encodes a protein crucial for cell adhesion.

6. Cowden Syndrome (CS): CS stems from mutations in the PTEN gene and is typified by multiple noncancerous hamartomas, alongside heightened risks of developing diverse cancers, such as breast, thyroid, and endometrial cancers.

7. Hereditary Retinoblastoma (RB1): Hereditary retinoblastoma arises from mutations in the RB1 gene, leading to the formation of cancerous tumors in the retina, typically emerging in early childhood.

8. Von Hippel-Lindau Syndrome (VHL): VHL arises from mutations in the VHL gene and is characterized by the development of tumors and cysts in various organs, including the kidneys, adrenal glands, pancreas, and central nervous system.

If you have a family history of cancer, you should undergo regular cancer screenings for the early detection and management of cancer. You can undergo a regular full body health checkup at Jaslok Hospital Mumbai, which is one of the best hospitals in Mumbai for cancer screening and treatment.

#cancer #hereditary cancer #hereditary cancer syndrome #Lynch syndrome #inherited breast and ovarian cancer syndrome #familial adenomatous polyposis #full body health checkup #regular health checkups #cancer screening #health checkup packages

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thunderon · 12 days

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really wish i smoked cigarettes because i have many days that really call for me to be heavily leaning against a wall and dejectedly smoking a cigarette with a 1000yd stare

#alas i do not smoke #hereditary cancer folks #my chances are high enough without it #op

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dailycupofcreativitea · 9 months

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Omg guys so YEEAARRSS ago, when I was 12, my older sister who passed away from colon cancer (diagnosed at 17) got a genetic test done to see if it was hereditary.

Unrelated to my family history of cancer (i.e. motivated purely by fun interest), I got a career as a bioinformatician involved in cancer genome interpretation.

I only heard about this genetic test recently (I'm 25 now), so I asked my mom if she still had the results. And weirdly, I understood them, because the same stuff they were looking for over a decade ago (ex. microsatellite instability status) are stuff we report about tumours at my job.

It's so weird to think that those results were generated when I was 12, and I grew up and (unrelated) pursued a career that eventually led me to understand them 😳

(Also the results were that it's not hereditary, thank goodness. Make sure y'all watch your gut health closely!)

#musings #i am actually going to a conference on cancer genomics literally tomorrow #and attending a workshop about hereditary cancer testing #wild

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female-malice · 1 year

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When this research is covered by the media, you’ll almost never see an article suggest that we should change the system to get to the root of the problem. The advice is usually of the discrete, individualistic, consumer variety: buy organic, do more exercise, get more sleep, get tested earlier.

How about changing the system to ensure that the only vegetables we can eat are organic. Or change it to give people more free time to exercise or sleep; or how about we stop plastic production or halt new fossil fuel production to move us out of a carcinogenic world? These stories never tell us that the causes of cancer are baked into industrial life and cannot be solved by individual behaviour change. I suppose “end fossil fuelled industrial capitalism” doesn’t fill people with agency. But it’s exactly what we need to do.

Cancer in young people (indeed cancer in general) is another of the structural crises of our time, playing out almost unseen in the background, but guaranteed to warp the future. This crisis will further strain our world as we have known it, working alongside covid to create sicker societies. This should be an all-hands-on-deck period of modern human history. Governments should be mobilising to rapidly reconfigure society to prioritise human health, wellbeing and planetary flourishing. They won’t do it. But the good news is that what is unsustainable cannot be sustained. Change will come.

#cc #i read this article last night and i got so angry about the world i started crying #my brother had lymphoma. it was hereditary and both my dad and uncle had it too #but my dad and uncle just lived normal lives after recovering from lymphoma. they had no post-cancer problems #my brother on the other hand went from marathon runner to seriously disabled #he can't work. he has enough energy for short visits and that's it #and as with most post-cancer complications... there's no answers. he just has chronic illness for life now #my uncle's lymphoma treatment was in the 1970s. My dad's lymphoma treatment was in the 2000s. And my brother's treatment was in the 2010s #why did my brother have the worst outcome of the three of them?

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pointlessfrog · 4 months

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I’ve been relistening to the lectures a medical oncologist did a couple months ago in my uni course and his voice was so hot.. I’m giggling and twirling my hair

#his charisma… yes please tell me more about hereditary cancer #tbh the only competent lecturer from that course #uni tag

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godmodebeginswithlesbians · 1 year

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sick again

#probably more sick than i thought #need to schedule a doctors appointment #mia's mumblings #no real strength left these past couple days #+ chest pain #+ lump #+ hereditary breast cancer both sides of the family

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mihai-florescu · 2 years

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Oh lmao i reached tag limit

#ok lets recap #grandma with brain surgery that is already past the time the doctors gave her who is not mentally present but whenever she realises #shes losing her mind she starts saying she wants to die and my dad gets angry at her and says this is the fate god gave her #generally depressed other grandma who every time she talks to us brings up the war how much the world sucks and that she wants to die #and my mom tells her she cant blame the americans for everything and then they dont speak for 2 weeks #grandpa who died of cancer bc he didnt tell anyone he was sick and when he collapsed and they took him to the hospital #he died in less than a day (i was 2 years old so idk the details but im pretty sure thats when they found out he kept it from them)#other dead grandpa who was an alcoholic but im not sure exactly if he was sick or why he died #father who burries himself in work cuz it's also his hobby and most if not all his friends are ppl hes met through work so everything #kind of revolves around it #hereditary heart issues for which he's on multiple medications for life #overworked mother who is responsible for 2 countries finances who works 16 hrs a day with almost no breaks every day except weekends #am i missing something #ah yes none of these people admit theres anything wrong with them #and my dad helps and assesses phd candidates in his field and is part of the national academy #all their hopes and expectations are on me and im not allowed to show weakness or disappointment #theyve knows since i was little that theres something not quite right but theyre all too neurotic to really figure it out #they put me in therapy for a bit in 3rd grade for anxiety and stress #5th grade* but it didnt really do anything #which is why im saying i dont think aaany therapy is going to help aaaat all i think we should just die #ah yes brain tumor grandma? self discovered. all doctors told her she just has depression and shes hysterical cuz her husband cheated #but she was so good at her job (biology lab something something) that she proved to them she has a tumor. so yknow. very competent #hm i think this might be why my family just. doesnt really believe in depression and thinks you should just get over it #im tiredddd im sickkk of it all i cant live off of my parents forever cuz theyre overworking and burning out for their own future happiness #i cant live off of them #i dont even think they like me. they just love me as much as they need to (as long as i do everything im supposed to)#i remember after my first year of uni they had a family meeting being angry that i dont have all the credits #i didnt have them because the teachers just took longer to upload them. but otherwise i had passed everything. they didnt believe me #i cant even exist with them just the way i am i have to do hair removal surgery and ideally in their opinion plastic surgery to fit #a standard and make it easier to get a job. because pretty privilege

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latesthealthupdates · 5 months

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వంశపారంపర్యంగా వచ్చే క్యాన్సర్లు (hereditary-cancer-syndromes)

ఒకవేళ ఒక కుటుంబంలో కొన్ని తరాలుగా ఎవరో ఒకరికి క్యాన్సర్ వస్తుంది అంటే, ఇది యాదృచ్చికమా లేక సైంటిఫిక్ గా దీనికి ఏమైనా కారణం ఉందా అని సందేహం రావటం సహజమే. అలాగే ఒకవేళ సైంటిఫిక్ గా వంశపారంపర్యంగా క్యాన్సర్లు వస్తున్నాయంటే దీనిని నివారించడానికి వీలవుతుందో లేదో అనే భయం కూడా సహజమే. ఆ సందేహాలకు, భయాలకు తెరతీసేది అవగాహన మాత్రమే. వంశపారంపర్యంగా వచ్చే క్యాన్సర్లు మరియు వాటిని ఎలా ముందుగానే గుర్తించాలి అనే విషయాలను తెలుసుకోవటానికి ఇది పూర్తిగా చదవండి.

For more information visit: https://www.punarjanayurveda.com/hereditary-cancer-syndromes/

#cancer #hereditary-cancer-syndromes

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doctorjennifermelfi · 6 months

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i’ve been complaining about my job so much that it must have had some kind of monkey’s paw effect and now a team of 25 is being whittled down to 11 and absorbed by another company

#i’m being “moved to another department” loooool #this has been a hell of a month i knew something crazy would happen #oh yeah i also found out i might have a hereditary genetic mutation that makes it more likely for you to get every kind of cancer #but i won’t know for sure until 2025 probably!!#“why are you so happy and jolly” lmfao fucking hell #gonna go eat some bucatini about it

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oreenolivia968 · 6 months

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Do You Know Your Hereditary Risk for Breast Cancer? - Eileen West, MD and Associates

In today’s rapidly advancing field of women’s health medicine, I have been very pleased to see recent developments around breast cancer prevention. One such advancement that holds immense promise is genetic testing. Up to 10% of all breast cancer is caused by an inherited genetic mutation (1), and screening for high-risk family histories helps focus on early intervention. Genetic testing for breast cancer is a powerful tool that allows us to identify individuals who may be at higher risk for developing the disease due to specific genetic mutations, such as BRCA1 and BRCA2. https://www.eileenwestmd.com/cancer/do-you-know-your-hereditary-risk-for-breast-cancer/

#Hereditary Risk for Breast Cancer

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onealgraversen00 · 11 months

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Nf-κb Signaling In Tumor Pathways Focusing On Breast And Ovarian Cancer

ORR and a fifty seven per cent PSA response had been introduced. In sufferers without BRCA mutations, the tumour response price was solely eleven per cent and PSA response 6 per Many of those small molecules are able to restoring the expression of p16INK4a in various cancers. Genistein, an isoflavone current in soybean, is a cancer chemopreventive agent that inhibits cell proliferation and induces apoptosis (Gullett et al., 2010 ). Li et al. reported that genistein represses early breast tumorigenesis by way of epigenetic regulation of CDKN2A by impacting histone modifications as properly as by recruiting the BMI1-c-MYC complicated to the regulatory area in the CDKN2A promoter ( Li et al., 2013b ). In prostate cancer, treatment with genistein increased acetylated histones 3 and four of the CDKN2A transcription start sites ( Majid et al., 2008 ). Thus, soybean merchandise containing genistein could be helpful in preventing breast and prostate cancer by way of transcriptional activation of CDKN2A by modulating its epigenetic silencing ( Li et al., 2013b and Majid et al., 2008 ). Rajendran et al. reported that Nrf2 wildtype mice confirmed a better incidence of colon tumors than Nrf2 heterozygous (Nrf2+/− ) mice when handled with 1,2-dimethylhydrazine. Pancreatic screening in high-risk patients such as BRCA2 mutation carriers has been proposed to reduce PDAC-related mortality. In 2012, the International Cancer of the Pancreas Screening (CAPS) Consortium revealed pointers on the management of patients with elevated risk for familial PDAC (Canto et al. 2013). Just as a result of the genomic check doesn’t pick up a prostate cancer mutation does not mean you don’t have mutation/s that causes prostate cancer. advanced tubo-ovarian cancer. The current commonplace of care in the first-line setting for most sufferers with newly recognized superior tubo-ovarian cancer is platinum-taxane chemotherapy and cytoreductive surgery with a goal to remove all of the seen cancer. significant dose discount and delays. In HRD-positive metastatic pancreatic cancer, veliparib with FOLFOX (folinic acid, fluorouracil, and oxaliplatin) chemotherapy confirmed an ORR of fifty per cent, median PFS of seven.2 months, and median OS of eleven.1 months. In breast cancer, the section three BrighTNess trial randomly We have been actually anticipating one thing as his dad died of prostate cancer and his mom died of breast cancer....but nothing hereditary found. He then took the Guardant blood take a look at and a variety of other variants/mutations have been found, which expanded his therapy options. Reactivation of silenced CDKN2A or the inhibition of epigenetic repression of the gene could be a rational strategy for the prevention or remedy of varied cancers. In comparability with previous trials such because the practice-changing SOLO-1, which was carried out in a My cancer has PTEN misplaced which facilitates the copy of the cancer cells. FOXA1, Si-ZBP-89, Jmjd3, Mutant UHRF1 and c-JUN induce p16INK4a protein expression by re-activation of the CDKN2A promoter. Schematic structure of the INK4a /ARF locus and the function of p16INK4a in cells. of olaparib with bevacizumab as first-line upkeep remedy for advanced tubo-ovarian cancer, no matter BRCA1/2 mutation status. The Monte Carlo simulation of 1,000 patients confirmed that the cost and effectiveness within the PFS illness section have been $3,124 ± $1,534 and zero.905 ± 0.449 QALY for placebo and $139,563 ± $33,046 and 1.544 ± 0.408 QALY for maintenance olaparib. The cost-effectiveness acceptability curve confirmed a nearly 54% likelihood of maintenance olaparib and 46% probability of placebo being a cheap strategy at the threshold of $200,000 per further QALY gained (Figure 4). Furthermore, olaparib didn't significantly improve outcomes in cohort B that comprised different HRD mutations. The trials; PRIMA, PAOLA-1, and VELIA, offered at a Presidential Symposium on the Congress, provide evidence of improved survival for almost all affected person brca1 pancreatic cancer risk sub-types in high-grade superior tubo-ovarian cancer. The enhanced Warburg impact in addition to the elevated production of lactate in ATM-deficient CRPC cells prompted us to determine which enzyme within the glycolysis pathway is regulated by ATM deficiency. Actually, we examined the mRNA stage of LDHA in addition to different enzymes along the glycolysis pathway by RT-qPCR in the two cell strains. Only LDHA was dramatically upregulated in C4-2/ATM-KO cells (Fig. 4B), and this upregulation is further supported by the western blot displaying ~three-fold increase of LDHA protein expression in ATM-KO cells (Fig. 4C). To additional confirm the upregulation of LDHA by ATM loss, we handled C4-2 cells with ATM inhibitor KU and located that the expression of LDHA increased after 72-h therapy of cells with KU (Supplementary Fig. 2).

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lifesapolyp · 1 year

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Today begins Hereditary Colon Cancer Syndrome Awareness Week!

The syndrome I have is Familial Adenomatous Polyposis.

What syndrome do you have and what has your experience been with it?

#familial adenomatous polyposis #familial polyposis #colon cancer #cancer #hereditary colon cancer #awareness

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ceyhanmedya · 1 year

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What is Adenocarcinoma? What are the symptoms? 2023

New Post has been published on https://bankakredin.com/what-is-adenocarcinoma-what-are-the-symptoms-2023/

What is Adenocarcinoma?��What are the symptoms? 2023

Cells in the human body have the ability to divide, live and die under normal conditions. Sometimes, structural deterioration can be seen in cells for reasons that are scientifically intelligible or not. The resulting disruptions cause disruption in the division, growth and death cycle of cells. Adenocarcinoma, which is still one of the important health problems from the past years, is one of them. Adenocarcinomas, which are inherited or occur later with the lifestyle of the person, are diseases that occur in the organs (glandular organs) where the cells that secrete the secretion called mucus are located. Since mucus secretion is an important component of homeostasis in the body, adenocarcinomas can manifest in many places in the body.

Difference Between Cancer and Adenocarcinoma

Adenocarcinomas are formed by the rapid proliferation of mucus-secreting cells and the spread of the tumor to the outside of the organ. Sometimes, it can spread to the surrounding tissues and organs, even to organs far from the starting point of the tumor. The rapid and uncontrolled spread of cells in this way is called metastasis. Adenocarcinomas are a type of cancer in that they are found in secretory organs. In other words, not all cancers may be adenocarcinomas, but all adenocarcinomas are cancer types.

In Which Body Parts Are Adenocarcinoma Occurs?

Considering that approximately 70-75% of the human body is fluid and tissue, the number and importance of secretion-producing cells in the body can be understood. Types of adenocarcinoma can therefore be seen in different organs. The most common adenocarcinoma cases are seen in areas that produce dense mucus such as breast, pancreas, prostate, lung, colon, small intestine, stomach, esophagus, kidney, cervix. The rate of proliferation in the cells, the organ in which adenocarcinoma is seen, the lifestyle of the person, past diseases or hereditary characteristics determine the stage of development of the disease and the risk size.

Adenocarcinoma Findings According to the Organs It Is Seen

Each type of cancer is individual and may not require the same treatment. Likewise, the symptoms may not be the same in every patient. For example, lung adenocarcinoma and kidney adenocarcinoma do not show the same findings. Findings should be differentiated according to the places of involvement in the body:

Breast: It shows symptoms with a distinct mass that can be felt, inflammation and redness of the skin, and milk-like fluid coming out of the breast. Breast adenocarcinoma is the most common type of cancer in the breasts.

Pancreas: It shows symptoms with pain in the abdomen and back, fever, nausea and vomiting, weight loss. Patients often do not notice signs of pancreatic cancer early.

Prostate: This organ, located under the bladder in men, is the erectile gland with the most secretion content. Therefore, in the case of adenocarcinoma, the first symptoms are erection problems, bloody urine, frequent urge to urinate and difficult urination, pain in the lower abdomen, significant pain during ejaculation.

Lung: It shows symptoms with difficulty breathing, snoring, bloody sputum, chest pain, loss of taste, hoarseness, weight loss, chronic cough.

Colon and small intestine: Pain and gas in the lower abdomen, blood in the stool, constant desire to go to the toilet and not being able to defecate enough, irregularity in going to the toilet, prolonged diarrhea or constipation, fatigue and weight loss, vomiting, anemia, significant darkening in stool color. shows. Colon and small intestine adenocarcinomas usually present with similar findings.

Stomach: It shows symptoms with severe pain, vomiting, heartburn, bloating, difficult swallowing, and a feeling of fullness in the stomach after a few bites, in an area under the chest that can sometimes be confused with heart pain.

Esophagus: In this organ, which consists of approximately 20 cm long cartilage rings between the mouth and the stomach, the symptoms first appear with coughing, snoring, difficult swallowing, sore throat, coughing.

Kidney: When palpated by hand in the lateral and posterior abdomen, it presents with swelling, bloody urine, persistent pain, weight loss, anemia, and high fever.

Cervix: Symptoms of adenocarcinoma of the cervix, which forms the transition area between the uterus and the vagina, start with odorous discharge, abnormal bleeding during and after sexual intercourse, menstrual irregularities, bleeding seen in menopause, pain in the back and legs, and fatigue.

What Causes Adenocarcinoma?

There may be more than one factor that triggers the type of adenocarcinoma that occurs in any organ. There is a common misconception that all types of cancer are inherited. Not every cancer, or even more specialized type of adenocarcinoma, is inherited. Cancers can cause changes in a person’s genetics, but this is not the same as hereditary inheritance. However, among the common causes of all, the person’s lifestyle is the most common cause. The most common causes for mucinous adenocarcinoma can be listed:

Sedentary life

Obesity

Use of tobacco products

Extremely irregular and malnutrition

overexposure to radiation

Some viral and bacterial infections

Chemicals harmful to the human body

How Is Adenocarcinoma Diagnosed?

Growth on an organ or tissue is the first sign of metastasis. Sometimes, tumor masses that can be felt by hand are formed as a result of growth in the cells. The patient, who sees the symptoms and feels the enlarged tissue, is examined after applying to the clinic. Certain procedures are required for definitive diagnosis and diagnosis. In the first step, laboratory values are checked with hemogram, stool and urine tests. The location of the tumor mass is determined by imaging methods such as radiography, ultrasound, MR, CT, PET, and mammography. The sample taken from the determined mass and the biopsy result performed by the pathology department are awaited. Abnormal histological findings of the enlarged cell mass are observed with biopsy. It is an important step for definitive diagnosis. Different techniques can be used depending on where the biopsy sample will be taken. Generally, adenocarcinomas Since it occurs in the internal organs of the body, a small closed surgery method, for example, a few holes opened in the lower abdomen in a colon adenocarcinoma patient or the metastasized tissue with a sample taken from the rectum are examined. The diagnosis is made as a result of all examinations.

What Are Specific Treatments for Adenocarcinoma?

The fact that it is a patient-specific treatment rather than a disease provides more specific treatments in cancer solutions. Thanks to new developing technologies, treatment options increase every day. Among the most common treatment methods are methods such as:

Removing the tumor mass by surgical procedure,

Stopping the tumor tissue with radiotherapy,

Stopping metastasis thanks to the invasive drug taken with chemotherapy,

Ensuring phagocytosis of the growing tumor by stimulating the patient’s immune system with immunotherapy

Each treatment method is determined by a personalized system and the required doses. Individual treatments are formed for patients with the system of treating only the tumor mass with a point shot without damaging the tissues and organs without metastasis.

After Adenocarcinoma Treatment

Slowing down or stopping the metastasizing tissue is the first goal of treatment. While some patients can survive with the tissue or organ that continues to metastasize, some patients can recover from treatment by getting a definitive solution. The support of the patient’s relatives and physician during and after the treatment is as important as the treatment process. Side effects such as weight loss, loss of appetite, nausea, vomiting, fever, mouth sores, and hair loss may occur. In the treatment, it is not aimed to kill any tissue other than the tissue that metastasizes, but it is normal to see some side effects that are controlled with additional treatments.

Patients who are suspected or diagnosed with adenocarcinoma can request treatment with the technological facilities within the hospital by consulting the specialist physician.

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